basic local alignment search tool (blast) core nucleotide (core_nt) database Search Results


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basic local alignment search tool (blast) core nucleotide (core_nt) database  (Biotechnology Information)
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Basic Local Alignment Search Tool (Blast) Core Nucleotide (Core Nt) Database, supplied by Biotechnology Information, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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core_nt database  (Biotechnology Information)
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antibody corest 07-455  (Millipore)
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Downregulation <t>of</t> <t>DDX17</t> and DDX5 during neuronal differentiation. ( A ) Western-blot analysis showing the decreased expression of DDX17 and DDX5 during differentiation of SH-SY5Y cells. The pro-proliferative MYCN protein, the neural cell adhesion molecule (NCAM1) and the neuronal gene repressors REST and <t>CoREST</t> were used as controls of differentiation. A larger REST immunoblot is shown in . ( B ) Prediction of neuronal miRNA binding sites in the 3′ UTR of DDX17 transcripts (TargetScanHuman v6.2). The nucleotides deleted in the mutant DDX17-UTR construct are in bold. ( C ) Effect of overexpressed miR-26a/b and miR-9 on the expression of endogenous DDX17 in SH-SY5Y cells. The quantification of the experiment is shown at the bottom. The amount of DDX17 is represented as the mean intensity of the signal normalized to GAPDH level ± S.E.M. ( n = 4 independent experiments) * P -value < 0.05; ** P -value < 0.01 (Student's t test). The DDX17 signal corresponds to addition of both p72 and p82 bands. ( D ) Luciferase assays showing the effect of miR-26a and miR-26b on the expression of wild-type and mutant DDX17 3′ UTR reporters. Relative luciferase units data are represented as the mean values ± S.E.M. of four independent experiments, normalized to the control sample set to 1. * P -value < 0.05 (Student's t test).
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dls data analysis: the corenn method  (LS Instruments AG)
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Downregulation <t>of</t> <t>DDX17</t> and DDX5 during neuronal differentiation. ( A ) Western-blot analysis showing the decreased expression of DDX17 and DDX5 during differentiation of SH-SY5Y cells. The pro-proliferative MYCN protein, the neural cell adhesion molecule (NCAM1) and the neuronal gene repressors REST and <t>CoREST</t> were used as controls of differentiation. A larger REST immunoblot is shown in . ( B ) Prediction of neuronal miRNA binding sites in the 3′ UTR of DDX17 transcripts (TargetScanHuman v6.2). The nucleotides deleted in the mutant DDX17-UTR construct are in bold. ( C ) Effect of overexpressed miR-26a/b and miR-9 on the expression of endogenous DDX17 in SH-SY5Y cells. The quantification of the experiment is shown at the bottom. The amount of DDX17 is represented as the mean intensity of the signal normalized to GAPDH level ± S.E.M. ( n = 4 independent experiments) * P -value < 0.05; ** P -value < 0.01 (Student's t test). The DDX17 signal corresponds to addition of both p72 and p82 bands. ( D ) Luciferase assays showing the effect of miR-26a and miR-26b on the expression of wild-type and mutant DDX17 3′ UTR reporters. Relative luciferase units data are represented as the mean values ± S.E.M. of four independent experiments, normalized to the control sample set to 1. * P -value < 0.05 (Student's t test).
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ncbi corenr database  (Biotechnology Information)
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Downregulation <t>of</t> <t>DDX17</t> and DDX5 during neuronal differentiation. ( A ) Western-blot analysis showing the decreased expression of DDX17 and DDX5 during differentiation of SH-SY5Y cells. The pro-proliferative MYCN protein, the neural cell adhesion molecule (NCAM1) and the neuronal gene repressors REST and <t>CoREST</t> were used as controls of differentiation. A larger REST immunoblot is shown in . ( B ) Prediction of neuronal miRNA binding sites in the 3′ UTR of DDX17 transcripts (TargetScanHuman v6.2). The nucleotides deleted in the mutant DDX17-UTR construct are in bold. ( C ) Effect of overexpressed miR-26a/b and miR-9 on the expression of endogenous DDX17 in SH-SY5Y cells. The quantification of the experiment is shown at the bottom. The amount of DDX17 is represented as the mean intensity of the signal normalized to GAPDH level ± S.E.M. ( n = 4 independent experiments) * P -value < 0.05; ** P -value < 0.01 (Student's t test). The DDX17 signal corresponds to addition of both p72 and p82 bands. ( D ) Luciferase assays showing the effect of miR-26a and miR-26b on the expression of wild-type and mutant DDX17 3′ UTR reporters. Relative luciferase units data are represented as the mean values ± S.E.M. of four independent experiments, normalized to the control sample set to 1. * P -value < 0.05 (Student's t test).
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neurotracker tm core (nt) 3d software program  (CogniSens Inc)
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Linear regression analysis with <t> neurotracker </t> (NT) mean speed threshold.
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anti-corest  (Millipore)
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Linear regression analysis with <t> neurotracker </t> (NT) mean speed threshold.
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corenn method  (LS Instruments AG)
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Linear regression analysis with <t> neurotracker </t> (NT) mean speed threshold.
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mitotic deacetylase midac  (MIDAC Corporation)
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Linear regression analysis with <t> neurotracker </t> (NT) mean speed threshold.
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hdac complexes midac  (MIDAC Corporation)
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Linear regression analysis with <t> neurotracker </t> (NT) mean speed threshold.
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Image Search Results


Downregulation of DDX17 and DDX5 during neuronal differentiation. ( A ) Western-blot analysis showing the decreased expression of DDX17 and DDX5 during differentiation of SH-SY5Y cells. The pro-proliferative MYCN protein, the neural cell adhesion molecule (NCAM1) and the neuronal gene repressors REST and CoREST were used as controls of differentiation. A larger REST immunoblot is shown in . ( B ) Prediction of neuronal miRNA binding sites in the 3′ UTR of DDX17 transcripts (TargetScanHuman v6.2). The nucleotides deleted in the mutant DDX17-UTR construct are in bold. ( C ) Effect of overexpressed miR-26a/b and miR-9 on the expression of endogenous DDX17 in SH-SY5Y cells. The quantification of the experiment is shown at the bottom. The amount of DDX17 is represented as the mean intensity of the signal normalized to GAPDH level ± S.E.M. ( n = 4 independent experiments) * P -value < 0.05; ** P -value < 0.01 (Student's t test). The DDX17 signal corresponds to addition of both p72 and p82 bands. ( D ) Luciferase assays showing the effect of miR-26a and miR-26b on the expression of wild-type and mutant DDX17 3′ UTR reporters. Relative luciferase units data are represented as the mean values ± S.E.M. of four independent experiments, normalized to the control sample set to 1. * P -value < 0.05 (Student's t test).

Journal: Nucleic Acids Research

Article Title: The RNA helicase DDX17 controls the transcriptional activity of REST and the expression of proneural microRNAs in neuronal differentiation

doi: 10.1093/nar/gky545

Figure Lengend Snippet: Downregulation of DDX17 and DDX5 during neuronal differentiation. ( A ) Western-blot analysis showing the decreased expression of DDX17 and DDX5 during differentiation of SH-SY5Y cells. The pro-proliferative MYCN protein, the neural cell adhesion molecule (NCAM1) and the neuronal gene repressors REST and CoREST were used as controls of differentiation. A larger REST immunoblot is shown in . ( B ) Prediction of neuronal miRNA binding sites in the 3′ UTR of DDX17 transcripts (TargetScanHuman v6.2). The nucleotides deleted in the mutant DDX17-UTR construct are in bold. ( C ) Effect of overexpressed miR-26a/b and miR-9 on the expression of endogenous DDX17 in SH-SY5Y cells. The quantification of the experiment is shown at the bottom. The amount of DDX17 is represented as the mean intensity of the signal normalized to GAPDH level ± S.E.M. ( n = 4 independent experiments) * P -value < 0.05; ** P -value < 0.01 (Student's t test). The DDX17 signal corresponds to addition of both p72 and p82 bands. ( D ) Luciferase assays showing the effect of miR-26a and miR-26b on the expression of wild-type and mutant DDX17 3′ UTR reporters. Relative luciferase units data are represented as the mean values ± S.E.M. of four independent experiments, normalized to the control sample set to 1. * P -value < 0.05 (Student's t test).

Article Snippet: Primary antibodies used for Western-blotting: DDX5 (ab10261, Abcam), DDX17 (ab24601, Abcam), MYCN (sc-56729, SantaCruz), NCAM1 (ab18277, Abcam), REST (07-579 Millipore), CoREST (07-455 Millipore), Synapsin-1 (D12G5, Ozyme), Actin (sc-1616, SantaCruz) and GAPDH (sc-32233, SantaCruz).

Techniques: Western Blot, Expressing, Binding Assay, Mutagenesis, Construct, Luciferase, Control

Linear regression analysis with neurotracker (NT) mean speed threshold.

Journal: Frontiers in Nutrition

Article Title: The impact of nutrition on visual cognitive performance in the nutrition, vision, and cognition in sport study

doi: 10.3389/fnut.2023.1208890

Figure Lengend Snippet: Linear regression analysis with neurotracker (NT) mean speed threshold.

Article Snippet: The NeuroTracker TM CORE (NT) 3D software program from CogniSens Inc. is a single-task integrative perceptual cognitive training system that is equivalent to the 3D-MOT system.

Techniques:

Linear regression analysis with neurotracker (NT) maximum speed threshold.

Journal: Frontiers in Nutrition

Article Title: The impact of nutrition on visual cognitive performance in the nutrition, vision, and cognition in sport study

doi: 10.3389/fnut.2023.1208890

Figure Lengend Snippet: Linear regression analysis with neurotracker (NT) maximum speed threshold.

Article Snippet: The NeuroTracker TM CORE (NT) 3D software program from CogniSens Inc. is a single-task integrative perceptual cognitive training system that is equivalent to the 3D-MOT system.

Techniques: